FIGURE 3.
FIGURE 3.
Inhibition of 18F-FDG uptake in HUVECs by cytochalasin B. Cytochalasin B competitively inhibits 18F-FDG uptake with an IC50 of 0.16 μmol/L. Values are mean ± COV (% of mean) of 2 independent experiments performed in triplicate (n = 6). c = concentration.
In this issue
Related Articles
Cited By...
- Imaging transcriptomics: Convergent cellular, transcriptomic, and molecular neuroimaging signatures in the healthy adult human brain
- FDG Uptake by Prosthetic Arterial Grafts in Large Vessel Vasculitis Is Not Specific for Active Disease
- A Continuously Infused Microfluidic Radioassay System for the Characterization of Cellular Pharmacokinetics
- Correlation Between In Vivo 18F-FDG PET and Immunohistochemical Markers of Glucose Uptake and Metabolism in Pheochromocytoma and Paraganglioma
- What Can Be Seen by 18F-FDG PET in Atherosclerosis Imaging? The Effect of Foam Cell Formation on 18F-FDG Uptake to Macrophages In Vitro
- Imaging Atherosclerosis With F18-Fluorodeoxyglucose Positron Emission Tomography: What Are We Actually Seeing?
- Hypoxia But Not Inflammation Augments Glucose Uptake in Human Macrophages: Implications for Imaging Atherosclerosis With 18Fluorine-Labeled 2-Deoxy-D-Glucose Positron Emission Tomography
- Imaging Atherosclerotic Plaque Inflammation by Fluorodeoxyglucose With Positron Emission Tomography: Ready for Prime Time?
- Inflammation Imaging in Atherosclerosis
- Quantification of Inflammation Within Rabbit Atherosclerotic Plaques Using the Macrophage-Specific CT Contrast Agent N1177: A Comparison with 18F-FDG PET/CT and Histology
- Fibronectin Stimulates Endothelial Cell 18F-FDG Uptake Through Focal Adhesion Kinase-Mediated Phosphatidylinositol 3-Kinase/Akt Signaling
- In Vitro Studies on the Signal Transduction of Thyroidal Uptake of 18F-FDG and 131I-Iodide
- Consensus Recommendations for the Use of 18F-FDG PET as an Indicator of Therapeutic Response in Patients in National Cancer Institute Trials
- 18F-FDG Imaging: Pitfalls and Artifacts
- Nitric Oxide Stimulates 18F-FDG Uptake in Human Endothelial Cells Through Increased Hexokinase Activity and GLUT1 Expression
- Cellular Origin and Molecular Mechanisms of 18F-FDG Uptake: Is There a Contribution of the Endothelium?