227 Objectives: Myocardial infarction (MI) triggers a dynamic and organized infiltration of the
damaged region by inflammatory leukocytes. Macrophages contribute to wound healing …

Persistent inflammation following myocardial infarction (MI) precipitates adverse outcome
including acute ventricular rupture and chronic heart failure. Molecular imaging allows …

37 Objectives:: The severity and duration of inflammation after myocardial infarction (MI) is
thought to impact infarct size and contractile dysfunction. We aimed to non-invasively …

Abstract Funding Acknowledgements Type of funding sources: Public grant (s)–National
budget only. Main funding source (s): Deutsche Forschungsgemeinschaft (DFG) Introduction …

163 Objectives: Tissue inflammation after acute myocardial infarction (MI) affects
endogenous wound healing. It has emerged as a therapeutic target. The relationship …

The immune-fibrosis axis plays a critical role in cardiac remodeling after acute myocardial
infarction. Imaging approaches to monitor temporal inflammation and fibroblast activation in …

Acute myocardial infarction is one of the leading causes of death in the western world.
Despite major improvements in myocardial reperfusion with sophisticated percutaneous …

Macrophages play the critical role of clearing necrotic debris in the wound healing response
that follows myocardial infarction (MI). Two days after MI, we labeled macrophages in vivo …

Background: Unbalanced tissue inflammation early after myocardial infarction (MI) promotes
infarct expansion and late contractile dysfunction. Targeted anti-inflammatory therapies are …

Background: Optimal healing of the myocardium after myocardial infarction (MI) requires a
suitable degree of inflammation and its timely resolution, together with a well-orchestrated …