134 Objectives: Cardiac injury including myocardial infarction (MI) precipitates the activation
and transdifferentiation of quiescent cardiac fibroblasts, leading to replacement and reactive …

P917 Introduction: Activation of quiescent cardiac fibroblasts after myocardial infarction is a
critical contributor to adverse cardiac remodeling and heart failure. Activated fibroblasts …

Heart failure remains a major source of late morbidity and mortality after myocardial
infarction (MI). The temporospatial presence of activated fibroblasts in the injured …

Abstract Purpose of Review Cardiovascular disease features adverse fibrotic processes
within the myocardium, leading to contractile dysfunction. Activated cardiac fibroblasts play a …

P703 Introduction: Myocardial fibroblast activation plays a central role in outcomes post-
acute myocardial infarction (MI). Initially fibroblasts are activated to stabilize the infarcted …

3333 Introduction: Heart failure (HF) is a chronic and progressive clinical syndrome in which
the heart structure or function is abnormal due to multiple factors. Fibroblast activation …

P310 Introduction: Chronic heart failure (CHF) is a progressive clinical syndrome
characterized by abnormal heart structure or function caused by multiple factors. With the …

Aims The aim of this retrospective analysis was to examine the pattern of cardiac 68 Ga-
fibroblast-activation protein-α inhibitor (FAPI) uptake in patients after acute myocardial …

Our previous study has demonstrated the feasibility of noninvasive imaging of fibroblast
activation protein (FAP)-expression after myocardial infarction (MI) in MI-territory in a rat …

2298 Introduction: Activation of quiescent cardiac fibroblasts after myocardial infarction (MI)
is a critical contributor to adverse cardiac remodeling and heart failure development. Novel …