Advanced glycation endproducts co-localize with inducible nitric oxide synthase in Alzheimer's disease

…, HJ Lüth, W Deuther-Conrad, S Dukic-Stefanovic… - Brain research, 2001 - Elsevier
Advanced glycation endproducts (AGEs), protein-bound oxidation products of sugars, have
been shown to be involved in the pathophysiological processes of Alzheimer’s disease (AD). …

AGES in brain ageing: AGE-inhibitors as neuroprotective and anti-dementia drugs?

S Dukic-Stefanovic, R Schinzel, P Riederer, G Münch* - Biogerontology, 2001 - Springer
In Alzheimer's disease, age-related cellular changes such ascompromised energy production
and increased radical formation areworsened by the presence of AGEs as additional, AD …

Anti‐inflammatory antioxidants attenuate the expression of inducible nitric oxide synthase mediated by advanced glycation endproducts in murine microglia

A Wong, S DukicStefanovic… - European Journal of …, 2001 - Wiley Online Library
Advanced glycation endproducts (AGEs) accumulate on long‐lived protein deposits including
β‐amyloid plaques in Alzheimer's disease (AD). AGE‐modified amyloid deposits contain …

Signal transduction pathways in mouse microglia N‐11 cells activated by advanced glycation endproducts (AGEs)

S DukicStefanovic, J Gasic‐Milenkovic… - Journal of …, 2003 - Wiley Online Library
Deposition of cross‐linked insoluble protein aggregates such as amyloid plaques is characteristic
for Alzheimer's disease. Microglial activation by these extracullar deposits has been …

β‐Amyloid peptide potentiates inflammatory responses induced by lipopolysaccharide, interferon‐γ and 'advanced glycation endproducts' in a murine microglia cell …

J Gasic‐Milenkovic, S DukicStefanovic… - European Journal of …, 2003 - Wiley Online Library
β‐Amyloid (Aβ) plaques are characteristic hallmarks of Alzheimer's disease (AD). In AD, it has
been suggested that activation of microglial cells might be the link between Aβ deposition …

Microglial activation induces cell death, inhibits neurite outgrowth and causes neurite retraction of differentiated neuroblastoma cells

…, J Gasic-Milenkovic, S Dukic-Stefanovic… - Experimental brain …, 2003 - Springer
Activation of glial cells has been proposed to contribute to neuronal dysfunction and neuronal
cell death in Alzheimer's disease. In this study, we attempt to determine some of the effects …

[HTML][HTML] Improved in vivo PET imaging of the adenosine A2A receptor in the brain using [18F]FLUDA, a deuterated radiotracer with high metabolic stability

…, M Toussaint, R Teodoro, S Dukić-Stefanović… - European journal of …, 2021 - Springer
Purpose The adenosine A 2A receptor has emerged as a therapeutic target for multiple
diseases, and thus the non-invasive imaging of the expression or occupancy of the A 2A receptor …

[HTML][HTML] Radiosynthesis and biological evaluation of [18F]AG-120 for PET imaging of the mutant isocitrate dehydrogenase 1 in glioma

TH Lai, B Wenzel, S Dukić-Stefanović… - European Journal of …, 2024 - Springer
Glioma are clinically challenging tumors due to their location and invasiveness nature, which
often hinder complete surgical resection. The evaluation of the isocitrate dehydrogenase …

[HTML][HTML] The human Müller cell line MIO-M1 expresses opsins

…, E Ulbricht, K Rillich, S Dukic-Stefanovic… - Molecular …, 2011 - ncbi.nlm.nih.gov
Purpose To determine whether the human Müller cell line Moorfields/Institute of
Ophthalmology-Müller 1 (MIO-M1) expresses opsins. Methods The gene expression of opsins was …

Expression of aquaporins in the retina of diabetic rats

M Hollborn, S Dukic-Stefanovic, T Pannicke… - Current Eye …, 2011 - Taylor & Francis
Purpose/Aim: The development of retinal edema is the main reason of impaired vision in
non-proliferative diabetic retinopathy. Water transport through aquaporins (AQPs) has been …