A serial echocardiographic observation of acute heart injury associated with pheochromocytoma crisis
Section snippets
Case report
A 50-year-old woman was hospitalized at 8:30 am on September 23, 1995, with a headache, hyperhydrosis and a feeling of chest oppression. Her pulse was 78 beats/min. Her blood pressure was 216/100 mm Hg. An electrocardiogram showed sinus rhythm and marked ST segment depression (−2 mm) in leads V4 to V6, II, III and a Vf. An echocardiogram showed normal contraction of the left ventricle and a percent fractional shortening (%FS) of 33% (normal: 30 to 50%). The plasma adrenaline level was 33 200
Discussion
Pheochromocytoma may cause anatomic and functional cardiac abnormalities [1]. Various echocardiographic abnormalities have been observed, including systolic anterior movement, global hypokinesis [2]as well as hypokinesis of the base and the apex [3]. However, information on echocardiographic characteristics is limited because this tumor is rare [4].
There are no previous reports of serial echocardiography during a pheochromocytoma crisis and no previous studies have examined the course of
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2015, Annales de Cardiologie et d'AngeiologieStress-related cardiomyopathy, ventricular dysfunction, artery thrombosis: A hidden pheochromocytoma
2014, American Journal of Emergency MedicineCitation Excerpt :The literature indicates that the pathophysiology of TTC and pheochromocytoma-related cardiomyopathy is similar and mediated by catecholamines [3]. The left ventricular wall motion abnormality in patients with pheochromocytoma crisis is generally global, although apical-sparing and TTC-like wall motion abnormalities have been reported [4,5]. Thus, Bybee and Prasad [2] suggested a modified version of the diagnostic criteria for TTC (Table 3), in which the lack of proven pheochromocytoma does not appear.
Acute left ventricular dysfunction of severe scorpion envenomation is related to myocardial perfusion disturbance
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2006, Endocrinology and Metabolism Clinics of North AmericaCitation Excerpt :These symptoms are caused by catecholamines, which induce vasoconstriction of the coronary arteries while simultaneously increasing myocardial oxygen demand through stimulation of heart rate and cardiac contractility. The presentation and electrocardiographic changes, such as ST-segment elevation or depression [69,71,72], negative T-waves, and a prolonged QT-interval (present in 7%–35% of patients [40,73]), may resemble those of patients with myocardial ischemia or infarction due to heart disease; however, patients with pheochromocytoma may also have other symptoms due to catecholamine excess, such as severe hypertension or headache, profuse sweating, or intense pallor. A history of episodic attacks is even more helpful.
Catecholamine-induced cardiomyopathy and pheochromocytoma
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