Catecholamine cardiotoxicity

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References (136)

  • HumphreyS.M. et al.

    Catecholamine-depletion and the no-flow phenomenon in anoxic and ischemic rat hearts

    J Mol Cell Cardiol

    (1982)
  • JosephX. et al.

    Determinants of resistance to the cardiotoxicity of isoproterenol in rats

    Toxicol Appl Pharmacol

    (1983)
  • MaseriA. et al.

    ‘Variant’ angina: one aspect of a continuous spectrum of vasopastic myocardial ischemia. Pathogenetic mechanism, estimated incidence, clinical and coronarographic findings in 138 patients

    Am J Cardiol

    (1978)
  • MosingerB. et al.

    Heart infarction-like effect induced by natural catecholamines in vitro

    Exp Pathol

    (1977)
  • OpieL.H.

    Metabolism of the heart in health and disease

    Am Heart J

    (1969)
    OpieL.H.

    Metabolism of the heart in health and disease

    Am Heart J

    (1969)
  • OphL.H. et al.

    Adrenalin-induced ‘oxygen-wastage’ and enzyme release from working rat heart. Effects of calcium antagonism, β-blockade, nicotinic acid and coronary artery ligation

    J Mol Cell Cardiol

    (1979)
  • OstadalB. et al.

    Occlusion of coronary vessels after administration of isopenaline, adrenalin and noradrenalin

    Physiol Bohemoslow

    (1967)
  • RaabW.

    Myocardial electrolyte derangement: crucial feature of pluricausal, so-called coronary heart disease

    Ann NY Acad Sci

    (1969)
  • RaabW. et al.

    Catecholamine-induced myocardial hypoxia in the presence of impaired coronary dilatability independent of external cardiac work

    Am J Cardiol

    (1962)
  • RamosK. et al.

    Prevention by L(−) ascorbic acid of isoproterenol-induced cardiotoxicity in primary cultures of rat myocytes

    Toxicology

    (1983)
  • ReichenbachD. et al.

    Myofibrillar degeneration: a response of the myocardial cell to injury

    Arch Pathol

    (1968)
  • ReichenbachD.D. et al.

    Pathology of the heart in sudden cardiac death

    Am J Cardiol

    (1977)
  • RonaG.

    Cardiac adaptation to insult

  • RonaG. et al.

    Role of coronary no-flow, reflow phenomenon in myocardial injury

  • AlpertL.I. et al.

    Cardiomyopathy associated with pheochromocytoma

    Arch Path

    (1972)
  • AmelinA.Y. et al.

    Experimental infarction-like changes of the myocardium under isadrine (isopropyl noradrenalin)

    Latvian Acad Science News

    (1966)
  • BaroldiG.

    Human myocardial infarction: coronarogenic and noncoronarogenic coagulation necrosis?

  • BaroldiG. et al.

    Coronary circulation in the normal and the pathologic heart

    (1967)
  • BhagatB. et al.

    cAMP activity and isoproterenol-induced myocardial injury in rats

  • BlaiklockR.G. et al.

    Epinephrine-induced myocardial necrosis: effects of aminophyline and adrenergic blockade

    Res. Communications in Chem Path and Pharmacol

    (1981)
  • BloomS.

    Reversible and irreversible injury: calcium as a major determinant

  • BloomS. et al.

    Myocytolysis and mitochondrial calcification in rat myocardium after low doses of isoproterenol

    Am J Path

    (1969)
  • BloomS. et al.

    Calcium as mediator of isoproterenol-induced myocardial necrosis

    Am J Path

    (1972)
  • BoutetM. et al.

    Etude des lésions myocardiques provoquées par l'isoprotérénol à l'aide de traceurs de diffusion

    La vie médicale au Canada français

    (1972)
  • BoutetM. et al.

    Aspect microcirculatoire des lésions myocardiques provoquées par l'infusion de catécholamines. Etude ultrastructurale à l'aide de traceurs de diffusion. I. Isoprotérénol

    Path et Biol

    (1973)
  • BoutetM. et al.

    Evolution des lésions myocardiques provoquées par l'infusion de catécholamines. Etude à l'aide du traceur de diffusion peroxydase

    La vie médicale au Canada français

    (1973)
  • BoutetM. et al.

    Aspect microcirculatoire des lésions myocardiques provoquées par l'infusion de catécholamines. Etude ultrastructurale à l'aide de traceurs de diffusion. Norépinéphrine

    Path et Biol

    (1974)
  • BoutetM. et al.

    Permeability alteration of sarcolemmal membrane in catecholamine-induced cardiac muscle cell injury. In vitro studies with fine structural diffusion tracer horseradish peroxidase

    Lab Invest

    (1976)
  • BüchnerF.

    Die Rolle des Herzmuskels bei der Angina pectoris

    Beitr Path Anat

    (1932)
  • BüchnerF. et al.
  • ChappelC.I. et al.

    Comparison of cardiotoxic actions of certain sympathomimetic amines

    Canad J Biochem

    (1959)
  • ChappelC.I. et al.

    Severe myocardial necrosis produced by isoproterenol in the rat

    Arch. Int. Pharmacodyn. therapie

    (1959)
  • CorrP.B. et al.

    Increased alpha adrenergic receptors in ischemic cat myocardium

    J Clin Invest

    (1981)
  • CowanM.J. et al.

    Selective myocardial cell necrosis in non-human primates

    Arch Path Lab Med

    (1983)
  • CsapoZ. et al.

    Early alterations of the cardiac muscle cells in isoproterenol-induced necrosis

    Arch Path

    (1972)
  • DusekJ. et al.

    Myocardial resistance to isoprenaline in rats: variations with time

    J Pathol

    (1971)
  • EliotR.S. et al.

    Influence of environmental stress on pathogenesis of sudden cardiac death

    Fed Proc

    (1977)
  • FerransV.J. et al.

    Isoproterenol-induced myocardial necrosis. A histochemical and electron microscopic study

    Am Heart J

    (1964)
  • FerransV.J. et al.
  • FerransV.J. et al.

    Histochemical and electron microscopic studies on the cardiac necroses produced by sympathomimetic agents

    Ann NY Acad Sci

    (1969)
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      Isoproterenol [1-(3,4-dihydroxyphenyl)-2-isopropylaminoethanol hydrochloride] is a synthetic material with catecholamine property and β-adrenergic agonist has dual effects at different doses. It can create severe myocardial infraction at high doses and induce heart function improvement at low doses [2,3]. Myocardial necrosis was created after cardiac dysfunction at high dose Isoproterenol via increase peroxidation of myocardial lipids by alterations in cardiac enzymes and antioxidants [4,5].

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