Neurotransmitter receptors and monoamine metabolites in the brains of patients with Alzheimer-type dementia and depression, and suicides
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2018, Journal of Chemical NeuroanatomyEvaluation of 5-HT<inf>2A</inf> and mGlu<inf>2/3</inf> receptors in postmortem prefrontal cortex of subjects with major depressive disorder: Effect of antidepressant treatment
2014, NeuropharmacologyCitation Excerpt :Moreover, higher postmortem prefrontal 5-HT2R binding has also been shown to correlate with lifetime aggression in suicide (Oquendo et al., 2006). Nevertheless, other studies have not found significant differences in prefrontal cortex 5-HT2AR density neither between suicide and non-suicide subjects (Oquendo et al., 2006) nor between depressed suicide subjects and controls (Owen et al., 1983; Crow et al., 1984; Cheetham et al., 1988; Arranz et al., 1994; Lowther et al., 1994; Stockmeier et al., 1997; Rosel et al., 2000; Underwood et al., 2012). Additionally, we have previously reported that prefrontal cortex [3H]ketanserin binding did not differ in a group of suicide victims with a variety of psychiatric disorders as compared to controls (Muguruza et al., 2013).
Dysregulated 5-HT<inf>2A</inf> receptor binding in postmortem frontal cortex of schizophrenic subjects
2013, European NeuropsychopharmacologyCitation Excerpt :In this study, there were no differences in [3H]ketanserin binding to the 5-HT2AR in a group of suicide victims with psychiatric disorders such as dysthymic disorder, alcoholism, and anorexia nervosa, among others. Similarly, previous work has shown that [3H]ketanserin binding is unaffected in postmortem frontal cortex of suicide victims with major depression (Owen et al., 1983; Crow et al., 1984; Cheetham et al., 1988; Arranz et al., 1994; Lowther et al., 1994; Stockmeier et al., 1997; Rosel et al., 2000). Few studies using [3H]ketanserin reported that the 5-HT2AR is altered in suicide victims without psychiatric diagnosis (Gross-Isseroff et al., 1990a; Turecki et al., 1999). [