This is a PDF-only article. The first page of the PDF of this article appears above.
In this issue
Jump to section
Related Articles
Cited By...
- Clinical Quantification of Myocardial Blood Flow Using PET: Joint Position Paper of the SNMMI Cardiovascular Council and the ASNC
- Arterial CO2 as a Potent Coronary Vasodilator: A Preclinical PET/MR Validation Study with Implications for Cardiac Stress Testing
- Impact of Unexpected Factors on Quantitative Myocardial Perfusion and Coronary Flow Reserve in Young, Asymptomatic Volunteers
- Regadenoson: A New Myocardial Stress Agent
- High-Dose Adenosine Overcomes the Attenuation of Myocardial Perfusion Reserve Caused by Caffeine
- Impact of Type 2 Diabetes on Nitric Oxide and Adrenergic Modulation of Myocardial Perfusion
- Nuclear cardiology: myocardial perfusion imaging with SPECT and PET
- Effect of Caffeine on Ischemia Detection by Adenosine Single-Photon Emission Computed Tomography Perfusion Imaging
- Caffeine Decreases Exercise-Induced Myocardial Flow Reserve
- Effect of Caffeine Intake on Myocardial Hyperemic Flow Induced by Adenosine Triphosphate and Dipyridamole
- Procedure guidelines for radionuclide myocardial perfusion imaging
- Influence of propranolol, enalaprilat, verapamil, and caffeine on adenosine A2A-receptor-mediated coronary vasodilation
- Serum Caffeine Levels After 24-Hour Abstention: Clinical Implications on Dipyridamole 201Tl Myocardial Perfusion Imaging
- Peripheral Flow Response to Transient Arterial Forearm Occlusion Does Not Reflect Myocardial Perfusion Reserve
- Regional myocardial blood flow in patients with sick sinus syndrome randomized to long-term single chamber atrial or dual chamber pacing--effect of pacing mode and rate
- Endothelium-Dependent and -Independent Perfusion Reserve and the Effect of L-arginine on Myocardial Perfusion in Patients With Syndrome X
- Effects of Long-term Smoking on Myocardial Blood Flow, Coronary Vasomotion, and Vasodilator Capacity
- Impaired Myocardial Vasodilation During Hyperemic Stress With Dipyridamole in Hypertriglyceridemia
- Altered Myocardial Vasodilatation in Patients With Hypertriglyceridemia in Anatomically Normal Coronary Arteries
- Reduced Myocardial Flow Reserve in Non-Insulin-Dependent Diabetes Mellitus