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FIGURE 2. BMIPP scintigrams of vasospastic angina before and after medical treatment of 58-y-old man (patient 1 in group I). Patient had spontaneous vasospastic attacks, and coronary vasospasm was induced in LAD, LCX, and RCA by ergonovine provocation. Decreased BMIPP uptake was observed in anterior and inferior regions (BMIPP-1). TDS of BMIPP was 16. Decreased MIBG uptake was seen in anterior, lateral, inferior, and apex regions (MIBG-1). TDS of MIBG was 34. Although anginal attacks were completely suppressed after 6 mo of medical treatment, vasospasm was reinduced in 3 coronary arteries by ergonovine provocation. TDS and percentage improvement of BMIPP were 11 and 31%. TDS and percentage improvement of MIBG were 24 and 29% (BMIPP-2 and MIBG-2). Patient’s percentage improvements of both tracers were lower than those in group II (Fig. 1)
