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The Role of PET in Lymphoma*

Yuliya S. Jhanwar1 and David J. Straus2

1 Nuclear Medicine Service, Department of Radiology, Memorial Sloan-Kettering Cancer Center, New York, New York; and 2 Lymphoma Service, Division of Hematologic Oncology, Department of Medicine, Memorial Sloan-Kettering Cancer Center, New York, New York


Figure 1
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  		FIGURE 1.  Pretherapy (A) and follow-up posttherapy (B) PET scans of patient with Burkitt's lymphoma involving multiple vertebrae (arrows). Follow-up scan demonstrates diffuse bone marrow uptake secondary to administration of growth factor, with decreased uptake seen in areas of previous bone marrow involvement.

 

Figure 2
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  		FIGURE 2.  Pretherapy (A) and follow-up posttherapy (B) PET scans of patient with diffuse large B-cell lymphoma with extensive 18F-FDG–avid disease in the neck, mediastinum, and peritoneal infiltration. Follow-up scan demonstrates resolution of disease.

 

Figure 3
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  		FIGURE 3.  Pretherapy (A) and posttherapy (B and C) PET scans (left) and CT scans (right) of patient with diffuse large B-cell lymphoma and large anterior mediastinal/right pericardial mass. Pretherapy scan shows intense 18F-FDG uptake. One follow-up scan (B) demonstrates shrinkage of mass but persistent uptake. Biopsy was negative for malignant cells. A later follow-up scan (C) demonstrates growth of residual 18F-FDG–avid mass.

 

Figure 4
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  		FIGURE 4.  Summary of management of HD and aggressive NHL using 18F-FDG PET.

 




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