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Nitrate Administration Increases Blood Flow in Dysfunctional but Viable Myocardium, Leading to Improved Assessment of Myocardial Viability: A PET Study

Riemer H.J.A. Slart1, Ali Agool1, Dirk J. van Veldhuisen2, Rudi A. Dierckx1 and Jeroen J. Bax3

1 Department of Nuclear Medicine and Molecular Imaging, University Medical Center, Groningen, The Netherlands; 2 Department of Cardiology, University Medical Center, Groningen, The Netherlands; and 3 Department of Cardiology, University Medical Center, Leiden, The Netherlands


Figure 1
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  		FIGURE 1.  13N-Ammonia flow parameters in 18F-FDG viable and nonviable segments. Baseline = 13N-ammonia baseline flow; NTG = 13N-ammonia-NTG. *Not statistically significant, 13N-ammonia baseline flow in viable segments vs. in nonviable segments. #P = 0.009, 13N-ammonia nitrate-enhanced flow in viable segments vs. in nonviable segments.

 

Figure 2
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  		FIGURE 2.  ROC curves of 13N-ammonia baseline, 13N-ammonia-NTG, and ratio of 13N-ammonia-NTG to 13N-ammonia baseline to detect viability on 18F-FDG PET in all dysfunctional segments. Ratio = ratio of 13N-ammonia-NTG to 13N-ammonia baseline; NTG = 13N-ammonia-NTG; rest = 13N-ammonia baseline.

 

Figure 3
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  		FIGURE 3.  ROC curves of 13N-ammonia baseline, 13N-ammonia-NTG, and ratio of 13N-ammonia-NTG to 13N-ammonia baseline to detect viability on 18F-FDG PET in akinetic or dyskinetic segments. Ratio = ratio of 13N-ammonia-NTG to 13N-ammonia baseline; NTG = 13N-ammonia-NTG; rest = 13N-ammonia baseline.

 




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