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Delayed Contrast Enhancement and Perfusable Tissue Index in Hypertrophic Cardiomyopathy: Comparison Between Cardiac MRI and PET

Paul Knaapen, MD1, Willem G. van Dockum, MD1, Olga Bondarenko, MD1, Wouter E.M. Kok, MD, PhD1, Marco J.W. Götte, MD, PhD1, Ronald Boellaard, PhD2, Aernout M. Beek, MD1, Cees A. Visser, MD, PhD1, Albert C. van Rossum, MD, PhD1, Adriaan A. Lammertsma, PhD2 and Frans C. Visser, MD, PhD1

1 Department of Cardiology, VU University Medical Center, Amsterdam, The Netherlands
2 Department of Nuclear Medicine and PET Research, VU University Medical Center, Amsterdam, The Netherlands



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  		FIGURE 1. Midventricular short-axis view of a gadolinium delayed enhanced CMR image (A) of HCM patient. Note patchy hyperenhancement located at hypertrophied interventricular septum at junctions of septum and right ventricular free walls (arrows). (B) Anatomic tissue fraction image of same patient derived by PET.

 


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  		FIGURE 2. DCE (A) and PTI (B) for healthy control subjects, HCM patients, and MI patients. *P < 0.05 vs. control subjects and HCM patients.

 


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  		FIGURE 3. PTI for segments with (DCE+) and without (DCE–) hyperenhancement in HCM patients. PTI was significantly higher in areas with hyperenhancement. *P < 0.05 for DCE+ vs. DCE–.

 


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  		FIGURE 4. Scatter plots show relation between DCE and PTI in HCM patients (A) and MI patients (B).

 


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  		FIGURE 5. Scatter plots show relation between LVEF and DCE (A) and PTI (B) in HCM patients.

 


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  		FIGURE 6. Short-axis view of DCE image (A) and parametric PTF image (B) in patient with anterior MI. Area of DCE matches absence of water-perfusable tissue in PTF image, indicating scar tissue. (C and D) Same images, as in A and B in HCM patient, demonstrate presence of water-perfusable tissue in hyperenhanced septal area.

 




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